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J. Lipid Res.
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Originally published In Press as doi:10.1194/jlr.R800057-JLR200 on November 17, 2008

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Journal of Lipid Research, Vol. 50, S395-S399, April 2009
Copyright © 2009 by American Society for Biochemistry and Molecular Biology

Lipids in Health and Disease

Adipocyte metabolism and obesity

Alan D. Attie1,* and Philipp E. Scherer{dagger}

* Department of Biochemistry, University of Wisconsin, Madison, WI 53706
{dagger} Touchstone Diabetes Center, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX 75390-8549

The authors' research is supported by the National Institutes of Health Grants R01-DK 66539 and R01-DK 58037 (A.D.A.), and R01-DK55758 and R01-CA112023 (P.E.S.). A.D.A. and P.E.S. are also supported by Juvenile Diabetes Research Foundation Grant 17-2007-1026.

Published, JLR Papers in Press, November 17, 2008.

1 To whom correspondence should be addressed. e-mail: adattie{at}wisc.edu


ABSTRACT

Adipose tissue metabolism exerts a profound impact on whole-body metabolism. We review how fuel partitioning between adipocytes and other tissues affects insulin signaling pathways. We discuss the role of adipose tissue inflammation in adipocyte metabolism and whole-body insulin sensitivity. Finally, we mention the role of adipokines in autocrine and paracrine signaling.

Supplementary key words adiponectin • leptin • inflammation

Abbreviations: BCAA, branched chain amino acid; PPAR{gamma}, peroxisome proliferator-activated receptor {gamma}; UPR, unfolded protein response


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Related Webpages:

JLR 50th Anniversary Collections
Anniversary Collection:: Lipids in Health and Disease



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